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We describe a clinical syndrome of delayed dystonia in children subsequent to initial gastrointestinal symptoms and acute noninflammatory encephalopathy.The syndrome was caused by the ingestion of mildewed sugarcane containing the Arthrinium-produced mycotoxin 3-nitropropionic acid(3-NPA).In the severely affected patients,intoxication usually was heralded by coma,with dystonia appearing 7 to 40 days after recovery from the coma.The dystonia was manifested as choreoathetosis,torsion spasms,or painful paroxysmal spasms of the extremities and was neither progressive nor reversible.CTs of the dystonic patients consistently showed bilateral hypodensities in the lenticular nuclei.The pathogenesis of the selective lenticular lesions induced by 3-NPA is not yet clear. |
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CAT scan
CAT scan,abnormal
coma
dystonia
dystonia,delayed onset
encephalopathy
lenticular nucleus,lesion of
lenticular nucleus,lesion of,bilateral
movement disorder
MRI
MRI,abnormal
mycotoxin
neurotoxin
nitroproprionic acid
seizure
striatum,lesion of
sugarcane,mildewed
toxic encephalopathy
toxins,nervous system
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